The absolute worst possible dietary pattern of eating for a type II diabetics is the standard diet advice given by dietitians and doctors across the country for almost all type ll diabetic patients, helping to lock in an international epidemic of type II diabetes. They routinely tell individuals to snack in order to maintain their blood sugar and to “stoke” their metabolism with fuel. In normal health, when you haven’t eaten for three hours, insulin levels return to a baseline. Now your pancreas makes a different hormone called glucagon. This hormone tells your liver to release the sugar it has stored (glycogen) to sustain your blood sugar levels, and as it does this it turns on your liver’s fat burning system. Why would one be advise to eat six small portion meals per day? Thus, under the influence of glucagon your liver simultaneously uses sugar and fat to sustain your blood sugar a true fat burning time that helps to clear up stagnating levels of triglycerides in your blood.
Also keep in mind that the body operates on a 24 hour cycle, like the 8 hour work day then you have a shift change. The body’s work day begins with assimilation. This is where we fuel the body when ready with the proper quality nutrients to sustain most of your day, this should always be the most energizing meal of the day. On the second shift comes digestion, the period where food is being broken down and used as fuel for the body with any extra energy being stored for later use. Third shift the body enters its cleansing phase, this period is where the body prepares to eliminate waste. Most of the time the body’s cleansing phase takes place over night when the body is at rest and this is the main reason you tend to go the toilet first thing in the morning. Yet we are taught we should eat 3 meals per day breakfast, lunch and dinner. Removing the body from its natural cycle, forcing it to perform nine cycles per day. Yet we wonder why the body is breaking down so rapidly.
If you snack on anything surpassing thirty calories you will raise insulin, which automatically turns off glucagen, causing fat burning to stop, and blunts the use of sugar that has been stored in your liver. However, since you haven’t used the stored sugar in your liver, then insuiin can’t put more sugar back in your liver as it normally would, meaning it will readily turn blood sugar into fat (even if you snacked on something with no fat). You are supposed to get a snack between meals, but it’s supposed to come from your liver. Not from eating. The worst thing for leptin and insulin are eating between meals, eating large meals, eating low fiber, eating highly refined sugar or refined carbohydrates and not exercising. If you eat anything after dinner, especially a heavy dinner you make matters even worse, because now you reduce the optimal access into your stored fat during sleep, a prime opportunity to burn fat. When this system is abused and weight is gained, then fat begins to accumulate in excess in your liver. (one of the main reasons some people may wake up tired is eating heavy diner and eating later, now when the body should be cleansing it has to be digesting. Working overtime)
The fat clogs your liver metabolism and reduces the ability of sugar to store in your liver following a meal. This is liver insulin resistance caused by fatty build up. This means that you are much more likely to become hypoglycemic or low blood sugar between meals, as you don’t have enough sugar in your warehouse to use for blood sugar between meals. This same fatty liver problem also gets in the way of how glucagon would burn fat between meals, causing glucagon to synthesize sugar in an inappropriate and out of-control manner, making blood sugar go high even though you haven’t eaten. This is why diabetics wake up with very high fasting blood sugar levels. These are complicated metabolic problems that are more difficult to fix than simple case of insulin resistance. Furthermore, your pancreas starts to tune out leptin, meaning that leptin resistance is occurring at the level of beta cells and the beta cells aren’t getting the leptin message to stop making insulin in a timely manner. This causes extra insulin to be made, which excessively lowers blood sugar by turning sugar to fat, while simultaneously inducing hypoglycemia or low blood sugar symptoms. Not to mention all the overtime work the pancreas is doing for because of the problems that this sugar cause.